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Homonymous visual field loss without structural lesion on magnetic resonance imaging: Documented with positron emission tomography and diffusion tensor imaging

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dc.contributor.authorYang, Tae-Ho-
dc.contributor.authorOh, Sun-Young-
dc.contributor.authorKwak, Kichang-
dc.contributor.authorLee, Jong Min-
dc.date.accessioned2022-07-16T03:36:44Z-
dc.date.available2022-07-16T03:36:44Z-
dc.date.created2021-05-13-
dc.date.issued2014-08-
dc.identifier.issn0165-8107-
dc.identifier.urihttps://scholarworks.bwise.kr/hanyang/handle/2021.sw.hanyang/159364-
dc.description.abstractThe authors describe a 35-year-old man suffering from homonymous hemianopia after head trauma 4 years before but with negative magnetic resonance imaging (MRI) findings. Brain fluorine-18 fluorodeoxyglucose positron emission tomography (18FDG-PET) showed hypometabolism at the unilateral occipital lobe and crossed cerebellar hemisphere, and diffusion tensor imaging (DTI) revealed that the ipsilateral optic radiations were completely interrupted. The crossed cerebellar diaschisis (CCD) observed in the chronic stage of brain damage was caused by cerebellar suppression of the cerebral blood flow due to an involvement of the corticopontocerebellar tract. PET and DTI provide objective means for determining the relationship of functional deficits to head trauma, even in cases where the injury was sustained years prior to the evaluation.-
dc.language영어-
dc.language.isoen-
dc.publisherInforma Healthcare-
dc.titleHomonymous visual field loss without structural lesion on magnetic resonance imaging: Documented with positron emission tomography and diffusion tensor imaging-
dc.typeArticle-
dc.contributor.affiliatedAuthorLee, Jong Min-
dc.identifier.doi10.3109/01658107.2014.897733-
dc.identifier.scopusid2-s2.0-84904596151-
dc.identifier.bibliographicCitationNeuro-Ophthalmology, v.38, no.4, pp.238 - 242-
dc.relation.isPartOfNeuro-Ophthalmology-
dc.citation.titleNeuro-Ophthalmology-
dc.citation.volume38-
dc.citation.number4-
dc.citation.startPage238-
dc.citation.endPage242-
dc.type.rimsART-
dc.type.docTypeArticle-
dc.description.journalClass1-
dc.description.isOpenAccessY-
dc.description.journalRegisteredClassscie-
dc.description.journalRegisteredClassscopus-
dc.subject.keywordPlusfluorodeoxyglucose f 18-
dc.subject.keywordPlusadult-
dc.subject.keywordPlusarticle-
dc.subject.keywordPlusataxia-
dc.subject.keywordPlusbrain blood flow-
dc.subject.keywordPlusbrain damage-
dc.subject.keywordPlusbrain disease-
dc.subject.keywordPluscase report-
dc.subject.keywordPluscrossed cerebellar diaschisis-
dc.subject.keywordPlusdiffusion tensor imaging-
dc.subject.keywordPlushomonymous hemianopia-
dc.subject.keywordPlushuman-
dc.subject.keywordPluslateral geniculate body-
dc.subject.keywordPlusmale-
dc.subject.keywordPlusmiddle cerebellar peduncle-
dc.subject.keywordPlusneuroimaging-
dc.subject.keywordPlusoccipital lobe-
dc.subject.keywordPluspositron emission tomography-
dc.subject.keywordPlustraumatic brain injury-
dc.subject.keywordPlusvisual field-
dc.subject.keywordAuthorCrossed cerebellar diaschisis-
dc.subject.keywordAuthorDiffusion tensor imaging-
dc.subject.keywordAuthorHomonymous hemianopia-
dc.subject.keywordAuthorTraumatic brain injury-
dc.identifier.urlhttps://www.tandfonline.com/doi/full/10.3109/01658107.2014.897733-
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