TNF-alpha polymorphisms in Korean adult-onset Still's disease patients
DC Field | Value | Language |
---|---|---|
dc.contributor.author | Sung, Yoon-Kyoung | - |
dc.contributor.author | Seong, Sang-Seokg | - |
dc.contributor.author | Woo, Jin-Hyun | - |
dc.contributor.author | Lee, Myung-Ho | - |
dc.contributor.author | Chung, Won-Tae | - |
dc.contributor.author | Choe, Jeong-Yoon | - |
dc.contributor.author | Yoo, Dae-Hyun | - |
dc.date.accessioned | 2022-12-21T01:54:50Z | - |
dc.date.available | 2022-12-21T01:54:50Z | - |
dc.date.created | 2022-08-26 | - |
dc.date.issued | 2008-07 | - |
dc.identifier.issn | 1756-1841 | - |
dc.identifier.uri | https://scholarworks.bwise.kr/hanyang/handle/2021.sw.hanyang/178162 | - |
dc.description.abstract | Aim: To investigate the role of TNF-alpha promoter polymorphisms in the pathogenesis of Korean adult-onset Still's disease (AOSD) patients. Methods: DNA was isolated from blood samples collected from 58 patients with AOSD meeting Yamaguchi's criteria and 105 healthy controls. TNF-alpha promoter polymorphisms at positions -1013, -863, -857, -308 were analysed by polymerase chain reaction-restriction fragment length polymorphism method. Genotype, allele, and estimated haplotype frequencies were compared between the AOSD group and controls. Results: The genotype, allele distributions, and estimated haplotype frequencies of the TNF-alpha polymorphism between AOSD patients and controls were not significantly different. In addition, TNF-alpha polymorphisms did not appear to have an effect on the disease course of the Korean AOSD patients. Conclusion: Genetic polymorphisms of TNF-alpha do not play a significant role in the development or in their course of Korean AOSD patients. | - |
dc.language | 영어 | - |
dc.language.iso | en | - |
dc.publisher | WILEY | - |
dc.title | TNF-alpha polymorphisms in Korean adult-onset Still's disease patients | - |
dc.type | Article | - |
dc.contributor.affiliatedAuthor | Sung, Yoon-Kyoung | - |
dc.contributor.affiliatedAuthor | Yoo, Dae-Hyun | - |
dc.identifier.doi | 10.1111/j.1756-185X.2008.00351.x | - |
dc.identifier.scopusid | 2-s2.0-58049137453 | - |
dc.identifier.wosid | 000266391300010 | - |
dc.identifier.bibliographicCitation | INTERNATIONAL JOURNAL OF RHEUMATIC DISEASES, v.11, no.2, pp.155 - 162 | - |
dc.relation.isPartOf | INTERNATIONAL JOURNAL OF RHEUMATIC DISEASES | - |
dc.citation.title | INTERNATIONAL JOURNAL OF RHEUMATIC DISEASES | - |
dc.citation.volume | 11 | - |
dc.citation.number | 2 | - |
dc.citation.startPage | 155 | - |
dc.citation.endPage | 162 | - |
dc.type.rims | ART | - |
dc.type.docType | Article | - |
dc.description.journalClass | 1 | - |
dc.description.isOpenAccess | N | - |
dc.description.journalRegisteredClass | scie | - |
dc.description.journalRegisteredClass | scopus | - |
dc.relation.journalResearchArea | Rheumatology | - |
dc.relation.journalWebOfScienceCategory | Rheumatology | - |
dc.subject.keywordPlus | NECROSIS-FACTOR-ALPHA | - |
dc.subject.keywordPlus | SYSTEMIC-LUPUS-ERYTHEMATOSUS | - |
dc.subject.keywordPlus | PROMOTER POLYMORPHISMS | - |
dc.subject.keywordPlus | RHEUMATOID-ARTHRITIS | - |
dc.subject.keywordPlus | ANKYLOSING-SPONDYLITIS | - |
dc.subject.keywordPlus | GENE POLYMORPHISMS | - |
dc.subject.keywordPlus | ASSOCIATION | - |
dc.subject.keywordPlus | HLA | - |
dc.subject.keywordPlus | MANIFESTATIONS | - |
dc.subject.keywordPlus | SUSCEPTIBILITY | - |
dc.subject.keywordAuthor | adult-onset Still&apos | - |
dc.subject.keywordAuthor | s disease | - |
dc.subject.keywordAuthor | Korean | - |
dc.subject.keywordAuthor | polymorphism | - |
dc.subject.keywordAuthor | TNF-alpha | - |
dc.identifier.url | https://onlinelibrary.wiley.com/doi/10.1111/j.1756-185X.2008.00351.x | - |
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