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Effect of low sodium and high potassium diet on lowering blood pressure and cardiovascular events

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dc.contributor.authorKim, Byung Sik-
dc.contributor.authorYu, Mi-Yeon-
dc.contributor.authorShin, Jinho-
dc.date.accessioned2024-01-15T07:30:18Z-
dc.date.available2024-01-15T07:30:18Z-
dc.date.issued2024-01-
dc.identifier.issn2635-6325-
dc.identifier.issn2056-5909-
dc.identifier.urihttps://scholarworks.bwise.kr/hanyang/handle/2021.sw.hanyang/194508-
dc.description.abstractIncorporating aggressive lifestyle modifications along with antihypertensive medication therapy is a crucial treatment strategy to enhance the control rate of hypertension. Dietary modification is one of the important lifestyle interventions for hypertension, and it has been proven to have a clear effect. Among food ingredients, sodium and potassium have been found to have the strongest association with blood pressure. The blood pressure-lowering effect of a low sodium diet and a high potassium diet has been well established, especially in hypertensive population. A high intake of potassium, a key component of the Dietary Approaches to Stop Hypertension (DASH) diet, has also shown a favorable impact on the risk of cardiovascular events. Additionally, research conducted with robust measurement methods has shown cardiovascular benefits of low-sodium intake. In this review, we aim to discuss the evidence regarding the relationship between the low sodium and high potassium diet and blood pressure and cardiovascular events.-
dc.format.extent13-
dc.language영어-
dc.language.isoENG-
dc.publisherKorean Society of Hypertension | BioMed Central-
dc.titleEffect of low sodium and high potassium diet on lowering blood pressure and cardiovascular events-
dc.typeArticle-
dc.publisher.location대한민국-
dc.identifier.doi10.1186/s40885-023-00259-0-
dc.identifier.scopusid2-s2.0-85180946822-
dc.identifier.wosid001134229300002-
dc.identifier.bibliographicCitationClinical Hypertension, v.30, no.1, pp 1 - 13-
dc.citation.titleClinical Hypertension-
dc.citation.volume30-
dc.citation.number1-
dc.citation.startPage1-
dc.citation.endPage13-
dc.type.docTypeReview-
dc.identifier.kciidART003040676-
dc.description.isOpenAccessY-
dc.description.journalRegisteredClassscopus-
dc.description.journalRegisteredClassesci-
dc.description.journalRegisteredClasskci-
dc.relation.journalResearchAreaCardiovascular System & Cardiology-
dc.relation.journalWebOfScienceCategoryPeripheral Vascular Disease-
dc.subject.keywordPlusK+-INDUCED NATRIURESIS-
dc.subject.keywordPlusMODEST SALT REDUCTION-
dc.subject.keywordPlus24-H URINARY SODIUM-
dc.subject.keywordPlusCHLORIDE COTRANSPORTER-
dc.subject.keywordPlusSCIENTIFIC STATEMENT-
dc.subject.keywordPlusCL-COTRANSPORTER-
dc.subject.keywordPlusORAL POTASSIUM-
dc.subject.keywordPlusNCC ACTIVITY-
dc.subject.keywordPlusALL-CAUSE-
dc.subject.keywordPlusEXCRETION-
dc.subject.keywordAuthorSodium-
dc.subject.keywordAuthorPotassium-
dc.subject.keywordAuthorBlood pressure-
dc.subject.keywordAuthorHypertension-
dc.subject.keywordAuthorCardiovascular event-
dc.identifier.urlhttps://clinicalhypertension.biomedcentral.com/articles/10.1186/s40885-023-00259-0-
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