Genetic association analysis of CNR1 and CNR2 polymorphisms with schizophrenia in a Korean population
DC Field | Value | Language |
---|---|---|
dc.contributor.author | Bae, Joon Seol | - |
dc.contributor.author | Kim, Jason Yongha | - |
dc.contributor.author | Park, Byung-Lae | - |
dc.contributor.author | Kim, Jeong-Hyun | - |
dc.contributor.author | Kim, Bomi | - |
dc.contributor.author | Park, Chul Soo | - |
dc.contributor.author | Kim, Bong-Jo | - |
dc.contributor.author | Lee, Cheol-Soon | - |
dc.contributor.author | Lee, Migyung | - |
dc.contributor.author | Choi, Woo Hyuk | - |
dc.contributor.author | Shin, Tae-Min | - |
dc.contributor.author | Hwang, Jaeuk | - |
dc.contributor.author | Shin, Hyoung Doo | - |
dc.contributor.author | Woo, Sung-Il | - |
dc.date.accessioned | 2021-08-11T22:25:17Z | - |
dc.date.available | 2021-08-11T22:25:17Z | - |
dc.date.issued | 2014-10 | - |
dc.identifier.issn | 0955-8829 | - |
dc.identifier.issn | 1473-5873 | - |
dc.identifier.uri | https://scholarworks.bwise.kr/sch/handle/2021.sw.sch/11830 | - |
dc.description.abstract | Located on 6q15 and 1p36.11, cannabinoid receptor 1 (CNR1) and cannabinoid receptor 2 (CNR2) genes are considered to be a positional and functional candidate gene for the development of mental disorders such as schizophrenia because CNR1 is known as a regulator of dopamine signaling in the hippocampus and the cerebral cortex. However, few genetic studies have been carried out to investigate an association of CNR1 and CNR2 polymorphisms and the risk of schizophrenia. In this study, although the result indicates that CNR1 and CNR2 variations are unlikely to influence schizophrenia susceptibility in a Korean population, the findings would provide meaningful information for further genetic studies. (C) 2014 Wolters Kluwer Health vertical bar Lippincott Williams & Wilkins. | - |
dc.format.extent | 5 | - |
dc.language | 영어 | - |
dc.language.iso | ENG | - |
dc.publisher | Lippincott Williams & Wilkins Ltd. | - |
dc.title | Genetic association analysis of CNR1 and CNR2 polymorphisms with schizophrenia in a Korean population | - |
dc.type | Article | - |
dc.publisher.location | 미국 | - |
dc.identifier.doi | 10.1097/YPG.0000000000000047 | - |
dc.identifier.scopusid | 2-s2.0-85027951686 | - |
dc.identifier.wosid | 000341507300007 | - |
dc.identifier.bibliographicCitation | Psychiatric Genetics, v.24, no.5, pp 225 - 229 | - |
dc.citation.title | Psychiatric Genetics | - |
dc.citation.volume | 24 | - |
dc.citation.number | 5 | - |
dc.citation.startPage | 225 | - |
dc.citation.endPage | 229 | - |
dc.type.docType | Article | - |
dc.description.isOpenAccess | N | - |
dc.description.journalRegisteredClass | scie | - |
dc.description.journalRegisteredClass | scopus | - |
dc.relation.journalResearchArea | Genetics & Heredity | - |
dc.relation.journalResearchArea | Neurosciences & Neurology | - |
dc.relation.journalWebOfScienceCategory | Genetics & Heredity | - |
dc.relation.journalWebOfScienceCategory | Neurosciences | - |
dc.subject.keywordPlus | CANNABINOID RECEPTOR GENE | - |
dc.subject.keywordPlus | CHINESE POPULATION | - |
dc.subject.keywordPlus | NO ASSOCIATION | - |
dc.subject.keywordPlus | (AAT)N REPEAT | - |
dc.subject.keywordPlus | SUSCEPTIBILITY | - |
dc.subject.keywordPlus | DEPENDENCE | - |
dc.subject.keywordPlus | PHARMACOLOGY | - |
dc.subject.keywordPlus | PHYSIOLOGY | - |
dc.subject.keywordPlus | MICRORNAS | - |
dc.subject.keywordPlus | MEMORY | - |
dc.subject.keywordAuthor | CNR1 | - |
dc.subject.keywordAuthor | CNR2 | - |
dc.subject.keywordAuthor | schizophrenia | - |
dc.subject.keywordAuthor | single-nucleotide polymorphism | - |
Items in ScholarWorks are protected by copyright, with all rights reserved, unless otherwise indicated.
(31538) 22, Soonchunhyang-ro, Asan-si, Chungcheongnam-do, Republic of Korea+82-41-530-1114
COPYRIGHT 2021 by SOONCHUNHYANG UNIVERSITY ALL RIGHTS RESERVED.
Certain data included herein are derived from the © Web of Science of Clarivate Analytics. All rights reserved.
You may not copy or re-distribute this material in whole or in part without the prior written consent of Clarivate Analytics.