Clinical Characteristics and Outcomes of Renal Infarction
DC Field | Value | Language |
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dc.contributor.author | Oh, Yun Kuy | - |
dc.contributor.author | Yang, Chul Woo | - |
dc.contributor.author | Kim, Yong-Lim | - |
dc.contributor.author | Kang, Shin-Wook | - |
dc.contributor.author | Park, Cheol Whee | - |
dc.contributor.author | Kim, Yon Su | - |
dc.contributor.author | Lee, Eun Young | - |
dc.contributor.author | Han, Byoung Geun | - |
dc.contributor.author | Lee, Sang Ho | - |
dc.contributor.author | Kim, Su-Hyun | - |
dc.contributor.author | Lee, Hajeong | - |
dc.contributor.author | Lim, Chun Soo | - |
dc.date.accessioned | 2021-08-11T18:24:16Z | - |
dc.date.available | 2021-08-11T18:24:16Z | - |
dc.date.issued | 2016-02 | - |
dc.identifier.issn | 0272-6386 | - |
dc.identifier.issn | 1523-6838 | - |
dc.identifier.uri | https://scholarworks.bwise.kr/sch/handle/2021.sw.sch/9408 | - |
dc.description.abstract | Background: Renal infarction is a rare condition resulting from an acute disruption of renal blood flow, and the cause and outcome of renal infarction are not well established. Study Design: Case series. Setting & Participants: 438 patients with renal infarction in January 1993 to December 2013 at 9 hospitals in Korea were included. Renal infarction was defined by radiologic findings that included single or multiple wedge-shaped parenchymal perfusion defects in the kidney. Predictor: Causes of renal infarction included cardiogenic (n = 244 [55.7%]), renal artery injury (n = 33 [7.5%]), hypercoagulable (n = 29 [6.6%]), and idiopathic (n = 132 [30.1%]) factors. Outcomes: We used recurrence, acute kidney injury (AKI; defined as creatinine level increase >= 0.3 mg/dL within 48 hours or an increase to 150% of baseline level within 7 days during the sentinel hospitalization), new-onset estimated glomerular filtration rate (eGFR) < 60 mL/min/1.73 m(2) (for >3 months after renal infarction in the absence of a history of decreased eGFR), end-stage renal disease (ESRD; receiving hemodialysis or peritoneal dialysis because of irreversible kidney damage), and mortality as outcome metrics. Results: Treatment included urokinase (n = 19), heparin (n = 342), warfarin (n = 330), and antiplatelet agents (n = 157). 5% of patients died during the initial hospitalization. During the median 20.0 (range, 1-223) months of follow-up, 2.8% of patients had recurrent infarction, 20.1% of patients developed AKI, 10.9% of patients developed new-onset eGFR, 60 mL/min/1.73 m(2), and 2.1% of patients progressed to ESRD. Limitations: This was a retrospective study; it cannot clearly determine the specific causal mechanism for certain patients or provide information about the causes of mortality. 16 patients were excluded from the prognostic analysis. Conclusions: Cardiogenic origins were the most important causes of renal infarction. Despite aggressive treatment, renal infarction can lead to AKI, new-onset eGFR < 60 mL/min/1.73 m(2), ESRD, and death. (C) 2016 by the National Kidney Foundation, Inc. | - |
dc.format.extent | 8 | - |
dc.language | 영어 | - |
dc.language.iso | ENG | - |
dc.publisher | W. B. Saunders Co., Ltd. | - |
dc.title | Clinical Characteristics and Outcomes of Renal Infarction | - |
dc.type | Article | - |
dc.publisher.location | 미국 | - |
dc.identifier.doi | 10.1053/j.ajkd.2015.09.019 | - |
dc.identifier.scopusid | 2-s2.0-84964989269 | - |
dc.identifier.wosid | 000368418800016 | - |
dc.identifier.bibliographicCitation | American Journal of Kidney Diseases, v.67, no.2, pp 243 - 250 | - |
dc.citation.title | American Journal of Kidney Diseases | - |
dc.citation.volume | 67 | - |
dc.citation.number | 2 | - |
dc.citation.startPage | 243 | - |
dc.citation.endPage | 250 | - |
dc.type.docType | Article | - |
dc.description.isOpenAccess | N | - |
dc.description.journalRegisteredClass | sci | - |
dc.description.journalRegisteredClass | scie | - |
dc.description.journalRegisteredClass | scopus | - |
dc.relation.journalResearchArea | Urology & Nephrology | - |
dc.relation.journalWebOfScienceCategory | Urology & Nephrology | - |
dc.subject.keywordPlus | GLOMERULAR-FILTRATION-RATE | - |
dc.subject.keywordPlus | DIAGNOSIS | - |
dc.subject.keywordPlus | EQUATION | - |
dc.subject.keywordPlus | EMBOLISM | - |
dc.subject.keywordPlus | DISEASE | - |
dc.subject.keywordPlus | DIET | - |
dc.subject.keywordPlus | CT | - |
dc.subject.keywordAuthor | Renal infarction | - |
dc.subject.keywordAuthor | parenchymal perfusion defect | - |
dc.subject.keywordAuthor | renal blood flow | - |
dc.subject.keywordAuthor | acute kidney injury (AKI) | - |
dc.subject.keywordAuthor | mortality | - |
dc.subject.keywordAuthor | kidney function | - |
dc.subject.keywordAuthor | reduced glomerular filtration rate | - |
dc.subject.keywordAuthor | end-stage renal disease (ESRD) | - |
dc.subject.keywordAuthor | cardiogenic etiology | - |
dc.subject.keywordAuthor | outcomes | - |
dc.subject.keywordAuthor | case series | - |
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