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Fluconazole-Resistant Candida parapsilosis Bloodstream Isolates with Y132F Mutation in ERG11 Gene, South Korea

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dc.contributor.authorChoi, YJ[Choi, Yong Jun]-
dc.contributor.authorKim, YJ[Kim, Yae-Jean]-
dc.contributor.authorYong, D[Yong, Dongeun]-
dc.contributor.authorByun, JH[Byun, Jung-Hyun]-
dc.contributor.authorKim, TS[Kim, Taek Soo]-
dc.contributor.authorChang, YS[Chang, Yun Sil]-
dc.contributor.authorChoi, MJ[Choi, Min Ji]-
dc.contributor.authorByeon, SA[Byeon, Seung Ah]-
dc.contributor.authorWon, EJ[Won, Eun Jeong]-
dc.contributor.authorKim, SH[Kim, Soo Hyun]-
dc.contributor.authorShin, MG[Shin, Myung Geun]-
dc.contributor.authorShin, JH[Shin, Jong Hee]-
dc.date.accessioned2021-07-29T11:25:32Z-
dc.date.available2021-07-29T11:25:32Z-
dc.date.created2018-12-18-
dc.date.issued2018-09-
dc.identifier.issn1080-6040-
dc.identifier.urihttps://scholarworks.bwise.kr/skku/handle/2021.sw.skku/18821-
dc.description.abstractWe recently observed the emergence of fluconazole-resistant Candida parapsilosis bloodstream isolates harboring a Y132F substitution in Erg11p in South Korea. These Y132F isolates had a higher propensity to cause clonal transmission than other fluconazole-resistant isolates and persisted within hospitals for several years, as revealed by microsatellite typing.-
dc.publisherCENTERS DISEASE CONTROL-
dc.subjectMOLECULAR-MECHANISMS-
dc.subjectAURIS-
dc.titleFluconazole-Resistant Candida parapsilosis Bloodstream Isolates with Y132F Mutation in ERG11 Gene, South Korea-
dc.typeArticle-
dc.contributor.affiliatedAuthorChang, YS[Chang, Yun Sil]-
dc.identifier.doi10.3201/eid2409.180625-
dc.identifier.scopusid2-s2.0-85052192895-
dc.identifier.wosid000441877400036-
dc.identifier.bibliographicCitationEMERGING INFECTIOUS DISEASES, v.24, no.9, pp.1768 - 1770-
dc.relation.isPartOfEMERGING INFECTIOUS DISEASES-
dc.citation.titleEMERGING INFECTIOUS DISEASES-
dc.citation.volume24-
dc.citation.number9-
dc.citation.startPage1768-
dc.citation.endPage1770-
dc.type.rimsART-
dc.type.docTypeLetter-
dc.description.journalClass1-
dc.description.isOpenAccessY-
dc.description.journalRegisteredClassscie-
dc.description.journalRegisteredClassscopus-
dc.relation.journalResearchAreaImmunology-
dc.relation.journalResearchAreaInfectious Diseases-
dc.relation.journalWebOfScienceCategoryImmunology-
dc.relation.journalWebOfScienceCategoryInfectious Diseases-
dc.subject.keywordPlusMOLECULAR-MECHANISMS-
dc.subject.keywordPlusAURIS-
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