Hyperlipidemia and erectile dysfunction
DC Field | Value | Language |
---|---|---|
dc.contributor.author | Kim, SC | - |
dc.date.accessioned | 2021-06-18T14:43:25Z | - |
dc.date.available | 2021-06-18T14:43:25Z | - |
dc.date.issued | 2000-09 | - |
dc.identifier.issn | 1008-682X | - |
dc.identifier.issn | 1745-7262 | - |
dc.identifier.uri | https://scholarworks.bwise.kr/cau/handle/2019.sw.cau/47351 | - |
dc.description.abstract | We have done consecutive studies to investigate the effects of impaired lipid metabolism on the contractile and relaxation response of cavernous smooth muscles and to elucidate its pathogenesis: 1) incidence of hyperlipidemia in impotent patients; 2) erection response to intracavernous injection of papaverine in impotent patients with hyperlipidemia; 3) relaxation responses of isolated cavernosal smooth muscles to endothelium-independent and endothelium-dependent vasodilators in impotent patients with hypercholesterolemia or hypertriglyceridemia; 4) involvement of superoxide radical in the impaired endothelium-dependent relaxation of cavernous smooth muscle in hypercholesterolemic rabbits; 5) effects of isolated lipoproteins and triglyceride, combined oxidized LDL plus triglyceride, and combined oxidized LDL plus HDL on contractile and relaxation response of rabbit cavernous smooth muscles; 6) involvement of e-NOS in the impaired endothelium-dependent relaxation of cavernous smooth muscle in hypercholesterolemic rabbit. Hypercholesterolemia may cause impairment of endothelium-dependent relaxation. Oxidized LDL is the major causative cholesterol of the impaired relaxation response. A chain reaction, the production of superoxide radicals and functional impairment of eNOS may be a major cause of the functional impairment in the early stages of hypercholesterolemia. | - |
dc.format.extent | 6 | - |
dc.language | 영어 | - |
dc.language.iso | ENG | - |
dc.publisher | SCIENCE PRESS | - |
dc.title | Hyperlipidemia and erectile dysfunction | - |
dc.type | Article | - |
dc.identifier.bibliographicCitation | ASIAN JOURNAL OF ANDROLOGY, v.2, no.3, pp 161 - 166 | - |
dc.description.isOpenAccess | N | - |
dc.identifier.wosid | 000165409900001 | - |
dc.identifier.scopusid | 2-s2.0-0034280812 | - |
dc.citation.endPage | 166 | - |
dc.citation.number | 3 | - |
dc.citation.startPage | 161 | - |
dc.citation.title | ASIAN JOURNAL OF ANDROLOGY | - |
dc.citation.volume | 2 | - |
dc.identifier.url | http://www.asiaandro.com/archive/1008-682X/2/161.htm | - |
dc.type.docType | Review | - |
dc.publisher.location | 중국 | - |
dc.subject.keywordAuthor | hypercholesterolemia | - |
dc.subject.keywordAuthor | hypertriglyceridemia | - |
dc.subject.keywordAuthor | LDL lipoproteins | - |
dc.subject.keywordAuthor | HDL lipoproteins | - |
dc.subject.keywordAuthor | superoxide radicals | - |
dc.subject.keywordAuthor | NO synthase | - |
dc.subject.keywordAuthor | cavernous smooth muscle | - |
dc.subject.keywordAuthor | erectile dysfunction | - |
dc.subject.keywordPlus | ENDOTHELIUM-DEPENDENT RELAXATION | - |
dc.subject.keywordPlus | LOW-DENSITY LIPOPROTEINS | - |
dc.subject.keywordPlus | CAVERNOUS SMOOTH-MUSCLE | - |
dc.subject.keywordPlus | NITRIC-OXIDE SYNTHASE | - |
dc.subject.keywordPlus | EXPERIMENTAL ATHEROSCLEROSIS | - |
dc.subject.keywordPlus | RABBIT | - |
dc.subject.keywordPlus | CHOLESTEROL | - |
dc.subject.keywordPlus | INVOLVEMENT | - |
dc.subject.keywordPlus | AORTA | - |
dc.relation.journalResearchArea | Endocrinology & Metabolism | - |
dc.relation.journalResearchArea | Urology & Nephrology | - |
dc.relation.journalWebOfScienceCategory | Andrology | - |
dc.relation.journalWebOfScienceCategory | Urology & Nephrology | - |
dc.description.journalRegisteredClass | scie | - |
dc.description.journalRegisteredClass | scopus | - |
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